Nellie Brutocao is a partner-elect in the technology transactions practice at Wilson Sonsini Goodrich & Rosati. Her practice focuses on the life sciences industry, representing public and private companies in intellectual property and commercial transactions, including strategic alliances, research and development collaborations, technology licenses and acquisitions, supply, distribution and manufacturing agreements, sponsored research agreements, clinical trials arrangements, material transfer agreements, and co-promotion arrangements. She has also represented life sciences companies in complex commercial and patent disputes, including Hatch-Waxman litigation.
During law school, Nellie clerked in the Civil Division of the U.S. Attorney's Office for the Northern District of California.
Prior to attending law school, Nellie worked at the University of California, San Francisco, where she conducted research and administered related experiments. Her work focused on brain receptors responsible for triggering early-onset psychosis.
Nellie Brutocao is a partner-elect in the technology transactions practice at Wilson Sonsini Goodrich & Rosati. Her practice focuses on the life sciences industry, representing public and private companies in intellectual property and commercial transactions, including strategic alliances, research and development collaborations, technology licenses and acquisitions, supply, distribution and manufacturing agreements, sponsored research agreements, clinical trials arrangements, material transfer agreements, and co-promotion arrangements. She has also represented life sciences companies in complex commercial and patent disputes, including Hatch-Waxman litigation.
During law school, Nellie clerked in the Civil Division of the U.S. Attorney's Office for the Northern District of California.
Prior to attending law school, Nellie worked at the University of California, San Francisco, where she conducted research and administered related experiments. Her work focused on brain receptors responsible for triggering early-onset psychosis.